IRIS
PAGANINI, CHIARA
 Distribuzione geografica
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Continente #
NA - Nord America 282
EU - Europa 195
AS - Asia 161
SA - Sud America 1
Totale 639
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Nazione #
US - Stati Uniti d'America 281
CN - Cina 114
IE - Irlanda 104
SG - Singapore 41
FI - Finlandia 37
DE - Germania 23
IT - Italia 22
SE - Svezia 4
JP - Giappone 3
IN - India 2
BR - Brasile 1
CA - Canada 1
FR - Francia 1
GB - Regno Unito 1
HK - Hong Kong 1
RS - Serbia 1
RU - Federazione Russa 1
UA - Ucraina 1
Totale 639
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Città #
Dublin 104
Chandler 69
Beijing 39
Helsinki 34
Singapore 32
Ashburn 23
Nanjing 23
Boardman 16
New York 16
Lawrence 13
Princeton 13
Medford 12
Wilmington 12
Seattle 9
Shanghai 9
Shenyang 7
Hangzhou 6
Houston 6
Jacksonville 6
Nanchang 6
Changsha 5
Los Angeles 5
Pavia 5
Pescara 5
Tianjin 5
Hebei 4
Munich 4
San Diego 4
Woodbridge 4
Jiaxing 3
Tokyo 3
Ann Arbor 2
Cambridge 2
Ergolding 2
Fairfield 2
Falkenstein 2
Maranello 2
Milan 2
Nuremberg 2
Piscataway 2
Pune 2
Redmond 2
Redwood City 2
San Francisco 2
Washington 2
Belgrade 1
Chengdu 1
Guangzhou 1
Hong Kong 1
Horben 1
London 1
Ningbo 1
Norwalk 1
Orange 1
Parma 1
Qingdao 1
San Genesio Ed Uniti 1
São Paulo 1
Toronto 1
Totale 544
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Nome #
N-acetylcysteine treatment ameliorates the skeletal phenotype of a mouse model of diastrophic dysplasia 64
Calcium activated nucleotidase 1 (CANT1) is critical for glycosaminoglycan biosynthesis in cartilage and endochondral ossification 62
Impaired proteoglycan glycosylation, elevated TGF-β signaling, and abnormal osteoblast differentiation as the basis for bone fragility in a mouse model for gerodermia osteodysplastica 60
Polyethylene Glycol-Poly-Lactide-co-Glycolide Block Copolymer-Based Nanoparticles as a Potential Tool for Off-Label Use of N-Acetylcysteine in the Treatment of Diastrophic Dysplasia 55
Bone and connective tissue disorders caused by defects in glycosaminoglycan biosynthesis: a panoramic view 46
Improvement of the skeletal phenotype in a mouse model of diastrophic dysplasia after postnatal treatment with N-acetylcysteine 43
Enzyme replacement therapy in mice lacking arylsulfatase B targets bone-remodeling cells, but not chondrocytes 41
Skeletal Dysplasias Caused by Sulfation Defects 40
Analysis of proteoglycan synthesis and secretion in cell culture systems 39
The Lysosomal Protein Arylsulfatase B Is a Key Enzyme Involved in Skeletal Turnover 38
Testing the Cre-mediated genetic switch for the generation of conditional knock-in mice 35
A Refractory Celiac Patient Successfully Treated With Mesenchymal Stem Cell Infusions 35
null 26
Phenotypic characterization of immortalized chondrocytes from a desbuquois dysplasia type 1 mouse model: A tool for studying defects in glycosaminoglycan biosynthesis 24
A Refractory Celiac Patient Successfully Treated With Mesenchymal Stem Cell Infusions 24
Identification of potential non-invasive biomarkers in diastrophic dysplasia 11
Cant1 Affects Cartilage Proteoglycan Properties: Aggrecan and Decorin Characterization in a Mouse Model of Desbuquois Dysplasia Type 1 11
Analysis of Aggrecan Glycanation by Western Blot in Cell Culture 7
Analysis of Aggrecan Glycanation by Western Blot in Cell Culture 5
Report on the 23rd FEBS Young Scientists’ Forum 2024 4
Biallelic variants in SLC35B2 cause a novel chondrodysplasia with hypomyelinating leukodystrophy 4
Biallelic variants in the SLC13A1 sulfate transporter gene cause hyposulfatemia with a mild spondylo‐epi‐metaphyseal dysplasia 4
Totale 678
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Categoria #
all - tutte 3.545
article - articoli 0
book - libri 0
conference - conferenze 0
curatela - curatele 0
other - altro 0
patent - brevetti 0
selected - selezionate 0
volume - volumi 0
Totale 3.545
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Totale Lug Ago Sett Ott Nov Dic Gen Feb Mar Apr Mag Giu
2019/202056 0 0 0 0 0 2 2 2 2 23 23 2
2020/202134 4 2 2 0 1 1 1 6 12 5 0 0
2021/202253 0 0 1 1 0 0 0 5 0 3 8 35
2022/2023220 25 26 0 8 10 18 0 12 111 3 6 1
2023/2024111 16 16 4 5 8 36 2 9 1 5 7 2
2024/2025106 5 16 3 24 17 41 0 0 0 0 0 0
Totale 678