The results of this in vitro study suggest that ethanol itself, not the ethanol metabolite acetaldehyde may be the primary agent responsible for developmental neurotoxic effects associated with heavy alcohol consumption.

Interaction of ethanol with muscarinic receptor-stimulated phosphoinositide during the brain growth spurt in the rat: role of acetaldehyde.

MANZO, LUIGI;
1993-01-01

Abstract

The results of this in vitro study suggest that ethanol itself, not the ethanol metabolite acetaldehyde may be the primary agent responsible for developmental neurotoxic effects associated with heavy alcohol consumption.
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Utilizza questo identificativo per citare o creare un link a questo documento: https://hdl.handle.net/11571/110909
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